TLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads.
dc.contributor.author | Low, Hui Zhi | |
dc.contributor.author | Ahrenstorf, Gerrit | |
dc.contributor.author | Pommerenke, Claudia | |
dc.contributor.author | Habermann, Nadine | |
dc.contributor.author | Schughart, Klaus | |
dc.contributor.author | Ordóñez, David | |
dc.contributor.author | Stripecke, Renata | |
dc.contributor.author | Wilk, Esther | |
dc.contributor.author | Witte, Torsten | |
dc.date.accessioned | 2016-04-20T08:00:47Z | en |
dc.date.available | 2016-04-20T08:00:47Z | en |
dc.date.issued | 2016 | en |
dc.identifier.citation | TLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads. 2016, 13 (1):15 Retrovirology | en |
dc.identifier.issn | 1742-4690 | en |
dc.identifier.pmid | 26969150 | en |
dc.identifier.doi | 10.1186/s12977-016-0248-y | en |
dc.identifier.uri | http://hdl.handle.net/10033/606041 | en |
dc.description.abstract | LILRA3 is an immunostimulatory molecule which can conditionally induce the proliferation of cytotoxic cells. LILRA3 has a deletion genotype which is associated with multiple immune disorders. In this study, we wanted to analyze the regulation of LILRA3 and its significance in the context of HIV infection. | |
dc.language.iso | en | en |
dc.title | TLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads. | en |
dc.type | Article | en |
dc.contributor.department | Department of Clinical Immunology and Rheumatology, Hannover Medical School, Carl-Neuberg-Str. 1, 30625, Hannover, Germany. | en |
dc.identifier.journal | Retrovirology | en |
refterms.dateFOA | 2018-06-12T17:48:13Z | |
html.description.abstract | LILRA3 is an immunostimulatory molecule which can conditionally induce the proliferation of cytotoxic cells. LILRA3 has a deletion genotype which is associated with multiple immune disorders. In this study, we wanted to analyze the regulation of LILRA3 and its significance in the context of HIV infection. |