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dc.contributor.authorLow, Hui Zhi
dc.contributor.authorAhrenstorf, Gerrit
dc.contributor.authorPommerenke, Claudia
dc.contributor.authorHabermann, Nadine
dc.contributor.authorSchughart, Klaus
dc.contributor.authorOrdóñez, David
dc.contributor.authorStripecke, Renata
dc.contributor.authorWilk, Esther
dc.contributor.authorWitte, Torsten
dc.date.accessioned2016-04-20T08:00:47Zen
dc.date.available2016-04-20T08:00:47Zen
dc.date.issued2016en
dc.identifier.citationTLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads. 2016, 13 (1):15 Retrovirologyen
dc.identifier.issn1742-4690en
dc.identifier.pmid26969150en
dc.identifier.doi10.1186/s12977-016-0248-yen
dc.identifier.urihttp://hdl.handle.net/10033/606041en
dc.description.abstractLILRA3 is an immunostimulatory molecule which can conditionally induce the proliferation of cytotoxic cells. LILRA3 has a deletion genotype which is associated with multiple immune disorders. In this study, we wanted to analyze the regulation of LILRA3 and its significance in the context of HIV infection.
dc.language.isoenen
dc.titleTLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads.en
dc.typeArticleen
dc.contributor.departmentDepartment of Clinical Immunology and Rheumatology, Hannover Medical School, Carl-Neuberg-Str. 1, 30625, Hannover, Germany.en
dc.identifier.journalRetrovirologyen
refterms.dateFOA2018-06-12T17:48:13Z
html.description.abstractLILRA3 is an immunostimulatory molecule which can conditionally induce the proliferation of cytotoxic cells. LILRA3 has a deletion genotype which is associated with multiple immune disorders. In this study, we wanted to analyze the regulation of LILRA3 and its significance in the context of HIV infection.


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